One of the most often recommended drug groups for hypertension (high blood pressure) and a basic treatment for heart failure are beta-blockers. Beta-Blocker reduce the heart’s need for oxygen by reducing the heart rate and suppressing the effects of epinephrine.
*High blood pressure
*Abnormal heart rhythms
Your doctor might not recommend a beta-blocker if you have asthma or COPD because it could exacerbate your breathing issues. Your doctor won’t recommend a beta-blocker if you have severe lung congestion in addition to heart failure.
They can be consumed in the morning, during meals, and before night. Due to your body absorbing the medication more slowly when you take it with meals, you may experience less side effects.
To find out how frequently to take it, refer to the label. Your condition will determine how often you need to take the drug, how long you may wait between doses, and how long you need to take it for. Usually, older individuals take smaller amounts. What should you do if you miss a dose? Ask your doctor.
There’s a chance you’ll need to monitor your pulse daily when using a beta-blocker. Contact your doctor about taking your beta-blocker that day if it’s slower than it should be.
Even if you believe a beta-blocker is not working, never stop taking it without first consulting your doctor. Heart attacks and angina can both be aggravated by abrupt cessation.
*Upset stomach, diarrhea, or constipation
*Shortness of breath
*Loss of sex drive or erectile dysfunction
If you have low blood pressure or a sluggish heartbeat, you shouldn’t take beta-blockers because doing so can make you feel faint and woozy by slowing your heart rate more.
Beta-blockers may have an adverse effect on a developing foetus by reducing blood pressure, blood sugar, and heart rate. These medications can also enter a baby through breast milk and result in low blood pressure, difficulty breathing, and a sluggish heartbeat.
If you are breastfeeding, attempting to get pregnant, or become pregnant while taking beta-blockers, you should let your doctor know.
Patients have a higher chance of developing heart failure after myocardial infarction (MI). Left ventricular systolic dysfunction in post-MI patients increases the risk of mortality or morbidity. Neuro-hormonal inhibitor pharmacological treatments for avoiding post-MI remodelling have been shown to significantly improve short- and long-term outcomes, including death, reinfarction, and worsening heart failure, according to strong and convincing findings from randomised studies. In treating post-MI patients with or without left ventricular systolic dysfunction (LVSD) and with or without heart failure symptoms and signs, proven pharmacological approaches are summarised in this article.
Studies have found that between 14 to 36% of individuals hospitalised for an acute MI also had HF. In the Global Registry of Acute Coronary Events (GRACE) study of 13,707 patients from 1999 to 2001, HF at admission increased the risk of in-hospital death by 2.2 times . 1,024 (13% of the total) deaths and 2,831 (37%) new HF diagnoses in MI patients occurred during the index MI hospitalisation. Hospital survivors (n = 4,291) who did not experience HF during their index hospitalisation had an additional 3,040 patients (71%) acquire HF by five years, with 64% of these cases occurring in the first year.
After a MI, there is a loss of cardiac muscle, which alters the impact zone and is ideally related to that. When the cardiac muscle is in its second stage, several molecular and metabolic processes are triggered, which result in ventricular dilatation hypertrophy and the development of scars. Over time, as this process continues, the preload and afterload rise, leading to ventricular remodelling. Best Cardiac Surgeon.